It is the ideal companion for both recreational flying and adventurous expeditions. Professors and postdoctoral fellows may apply for a CCK Scholar Grant ranging from $20,000 to $35,000 to help replace half of their salary while they’re on sabbatical, or for time off for research and writing. If grants from other sources are also awarded to the applicant, the Foundation’s grant, when c444ck added to these other grants, must not exceed the recipient’s annual salary. Priority will be given to collaborative projects involving institutions in Taiwan.
- Professors and postdoctoral fellows may apply for a CCK Scholar Grant ranging from $20,000 to $35,000 to help replace half of their salary while they’re on sabbatical, or for time off for research and writing.
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- The family also includes frog skin peptides (caerulein and phyllocaerulein) and the protochordean neuropeptide cionin, but in mammals, CCK and gastrin are the only family members (Figure 1).
Cholecystokinin (CCK) receptor family belongs to class-A sevenfold transmembrane G protein-coupled receptors, and is divided into CCK1 receptor (CCK1R) and CCK2 receptor (CCK2R)1. CCK1R is mainly distributed in the gastrointestinal tract2,3,4, peripheral nervous system5,6, and some regions of the brain, e.g., the area postrema, the nucleus tractus solitarius, and the hypothalamus7,8,9,10. Hence, CCK receptors (CCKRs) regulate a variety of physiological functions including digestion, satiety, emotion regulation, pain sensation, and memory process18,19,20,21,22,23,24,25. Besides, CCKRs are expressed in the brain–gut axis which is a region critical for the transmission of information between gut and brain including satiation signals. For these functions, there are already some specific drugs designed for CCK1R, such as ceruletide, or for CCK2R, including proglumide and pentagastrin. Although all of them have been used for gastrointestinal diseases, they and some drugs under clinical trial also have the potential to treat CNS diseases, including pain and anxiety26,27.
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But the last decades have uncovered additional sites and cell types that also express the CCK gene at peptide level (Table 1). In some of these sites, proCCK is not processed to the known α-amidated peptides. But since CCK receptors also have such widespread expression (66, 67, 70–73, 94, 95), there is both room and need for delineation of the roles of CCK released from the “new” sites. Also mentioned before (9), “CCK in circulation originates mainly from intestinal endocrine cells. The release to blood was not possible to examine until specific assays were developed (10–12, 54).
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